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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-269699
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.26969
Alternative Links zum Volltext:Pubmed
Zusammenfassung
In this study we have examined the subcellar pathways along which angiotensin II (ANG II) causes renal vasoconstriction. Using the isolated perfused rat kidney model, we found that renal vasoconstriction produced by ANG II (100 pM) was not altered by the calmodulin antagonists calmidazolium (1 microM) and N-(6-aminohexyl)-5-chloro-1-naphthalensulfonamide (W-7, 10 microM) but was blunted by ...
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