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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-309308
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.30930
Zusammenfassung
Introduction Osteoarthritis (OA) is characterized by an imbalance in cartilage and underlying subchondral bone homeostasis. We hypothesized that signals from the subchondral bone may modulate production of matrix components, alter chondrogenic differentiation potential of cocultured bone marrow-derived mesenchymal stem cells (BMSC) and induce a phenotypic shift in differentiated OA ...
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