Entries of Federlein, Anna on the publication server
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Number of items: 4.
Sinha, Frederick, Federlein, Anna, Biesold, Annika, Schwarzfischer, Magdalena, Krieger, Katharina, Schweda, Frank and Tauber, Philipp
(2023)
Empagliflozin increases kidney weight due to increased cell size in the proximal tubule S3 segment and the collecting duct.
Frontiers in Pharmacology 14.
Frees, Andreas, Assersen, Kasper B.
, Jensen, Mia, Hansen, Pernille B.L., Vanhoutte, Paul M., Madsen, Kirsten, Federlein, Anna, Lund, Lars
, Toft, Anja and Jensen, Boye L.
(2021)
Natriuretic peptides relax human intrarenal arteries through natriuretic peptide receptor type‐A recapitulated by soluble guanylyl cyclase agonists.
Acta Physiologica 231 (3).
Fulltext not available.
, Jensen, Mia, Hansen, Pernille B.L., Vanhoutte, Paul M., Madsen, Kirsten, Federlein, Anna, Lund, Lars
, Toft, Anja and Jensen, Boye L.
(2021)
Natriuretic peptides relax human intrarenal arteries through natriuretic peptide receptor type‐A recapitulated by soluble guanylyl cyclase agonists.
Acta Physiologica 231 (3).
Fulltext not available.
Steglich, Anne, Kessel, Friederike, Hickmann, Linda, Gerlach, Michael
, Lachmann, Peter, Gembardt, Florian, Lesche, Mathias, Dahl, Andreas
, Federlein, Anna, Schweda, Frank, Hugo, Christian P. M. and Todorov, Vladimir T.
(2019)
Renin cells with defective Gsα/cAMP signaling contribute to renal endothelial damage.
Pflügers Archiv - European Journal of Physiology 471 (9), pp. 1205-1217.
Fulltext not available.
, Lachmann, Peter, Gembardt, Florian, Lesche, Mathias, Dahl, Andreas
, Federlein, Anna, Schweda, Frank, Hugo, Christian P. M. and Todorov, Vladimir T.
(2019)
Renin cells with defective Gsα/cAMP signaling contribute to renal endothelial damage.
Pflügers Archiv - European Journal of Physiology 471 (9), pp. 1205-1217.
Fulltext not available.
Staffel, Janina, Valletta, Daniela, Federlein, Anna, Ehm, Katharina, Volkmann, Regine, Füchsl, Andrea M., Witzgall, Ralph, Kuhn, Michaela and Schweda, Frank
(2016)
Natriuretic Peptide Receptor Guanylyl Cyclase-A in Podocytes is Renoprotective but Dispensable for Physiologic Renal Function.
Journal of the American Society of Nephrology 28 (1), pp. 260-277.
Fulltext not available.
