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Human nephrosclerosis triggers a hypoxia-related glomerulopathy

Neusser, Matthias A., Lindenmeyer, Maja T., Moll, Anton G., Segerer, Stephan, Edenhofer, Ilka, Sen, Kontheari, Stiehl, Daniel P., Kretzler, Matthias, Gröne, Hermann-Josef, Schlöndorff, Detlef and Cohen, Clemens D. (2010) Human nephrosclerosis triggers a hypoxia-related glomerulopathy. The American journal of pathology 176 (2), pp. 594-607.

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Abstract

In the kidney, hypoxia contributes to tubulointerstitial fibrosis, but little is known about its implications for glomerular damage and glomerulosclerosis. Chronic hypoxia was hypothesized to be involved in nephrosclerosis (NSC) or "hypertensive nephropathy." In the present study genome-wide expression data from microdissected glomeruli were studied to examine the role of hypoxia in ...

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Item type:Article
Date:February 2010
Institutions:Medicine > Institut für Funktionelle Genomik > Lehrstuhl für Statistische Bioinformatik (Prof. Spang)
Identification Number:
ValueType
20019191PubMed ID
10.2353/ajpath.2010.090268DOI
Classification:
NotationType
AdolescentMESH
AdultMESH
AgedMESH
AnimalsMESH
Anoxia/geneticsMESH
Case-Control StudiesMESH
Cells, CulturedMESH
FemaleMESH
Gene Expression ProfilingMESH
HumansMESH
Kidney Diseases/pathologyMESH
Kidney Glomerulus/pathologyMESH
MaleMESH
MiceMESH
Middle AgedMESH
Nephrosclerosis/geneticsMESH
Oligonucleotide Array Sequence AnalysisMESH
Oxygen/pharmacologyMESH
Young AdultMESH
Dewey Decimal Classification:500 Science > 500 Natural sciences & mathematics
500 Science > 570 Life sciences
600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:No
Item ID:30642
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