| License: Creative Commons Attribution Non-commercial 4.0 PDF - Published Version (4MB) |
- URN to cite this document:
- urn:nbn:de:bvb:355-epub-516604
- DOI to cite this document:
- 10.5283/epub.51660
Abstract
Background PKARI alpha (protein kinase A type I-alpha regulatory subunit) is redox-active independent of its physiologic agonist cAMP. However, it is unknown whether this alternative mechanism of PKARI alpha activation may be of relevance to cardiac excitation-contraction coupling. Methods and Results We used a redox-dead transgenic mouse model with homozygous knock-in replacement of ...

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