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- URN to cite this document:
- urn:nbn:de:bvb:355-epub-269317
- DOI to cite this document:
- 10.5283/epub.26931
Alternative links to fulltext:Pubmed
Abstract
The results suggest that the suppression of renin gene expression in the contralateral kidneys of stenosed kidneys is not due to compensatory renal growth nor mediated by systemic blood pressure, angiotensin II AT1 receptors or renal nerves. We therefore hypothesize that kidneys with reduced perfusion release a humoral factor that acts as a potent inhibitor of renin gene expression.
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