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Role of protein kinase C in renal vasoconstriction caused by angiotensin II

Scholz, H. and Kurtz, Armin (1990) Role of protein kinase C in renal vasoconstriction caused by angiotensin II. The American journal of physiology. Cell physiology 259 (3 Pt 1), C421-C426.

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Abstract

In this study we have examined the subcellar pathways along which angiotensin II (ANG II) causes renal vasoconstriction. Using the isolated perfused rat kidney model, we found that renal vasoconstriction produced by ANG II (100 pM) was not altered by the calmodulin antagonists calmidazolium (1 microM) and N-(6-aminohexyl)-5-chloro-1-naphthalensulfonamide (W-7, 10 microM) but was blunted by ...

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Item type:Article
Date:1990
Institutions:Biology, Preclinical Medicine > Institut für Physiologie > Prof. Dr. Armin Kurtz
Identification Number:
ValueType
2399965PubMed ID
Classification:
NotationType
1-(5-Isoquinolinesulfonyl)-2-MethylpiperazineMESH
Alkaloids/pharmacologyMESH
Angiotensin II/pharmacologyMESH
AnimalsMESH
Cobalt/pharmacologyMESH
Enzyme Inhibitors/pharmacologyMESH
Imidazoles/pharmacologyMESH
Isoquinolines/pharmacologyMESH
MaleMESH
Nifedipine/pharmacologyMESH
Piperazines/pharmacologyMESH
Protein Kinase C/metabolismMESH
RatsMESH
Rats, Inbred StrainsMESH
Regional Blood Flow/drug effectsMESH
Renal Circulation/drug effectsMESH
StaurosporineMESH
Tetradecanoylphorbol Acetate/pharmacologyMESH
Vasoconstriction/drug effectsMESH
Verapamil/pharmacologyMESH
Dewey Decimal Classification:500 Science > 570 Life sciences
600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Unknown
Item ID:26969
Owner only: item control page

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