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- URN to cite this document:
- urn:nbn:de:bvb:355-epub-305265
- DOI to cite this document:
- 10.5283/epub.30526
Abstract
Human cancers almost ubiquitously harbor epigenetic alterations. Although such alterations in epigenetic marks, including DNA methylation, are potentially heritable, they can also be dynamically altered. Given this potential for plasticity, the degree to which epigenetic changes can be subject to selection and act as drivers of neoplasia has been questioned. We carried out genome-scale analyses ...

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