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The brain vasopressin system mediates maternal behaviour in lactating rats - impact of V1b receptors in hypothalamic and limbic brain regions

Bayerl, Doris (2017) The brain vasopressin system mediates maternal behaviour in lactating rats - impact of V1b receptors in hypothalamic and limbic brain regions. PhD, Universität Regensburg.

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Date of publication of this fulltext: 02 Jan 2017 11:21

Abstract (English)

Adequate maternal behaviour offers the best chance for the offspring to survive to maturity. This pro-social behaviour is known to be regulated by the nonapeptide arginine-vasopressin (AVP) amongst other peptidergic and non-peptidergic systems in rodents as well as in humans. Most research regarding the AVP system in maternal behaviour has focussed on the peptide itself and its V1a receptor ...

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Translation of the abstract (German)

Adäquates mütterliches Verhalten ermöglicht den Nachkommen die bestmögliche Chance bis zum Erwachsenenalter zu überleben. Dieses prosoziale Verhalten wird sowohl im Nager als auch im Menschen, unter anderem durch das Nonapeptid Arginin-Vasopressin (AVP), reguliert. Im Zusammenhang mit mütterlichem Verhalten lag der Forschungsschwerpunkt bezüglich des AVP-Systems bis jetzt auf dem Peptid selbst, ...

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Item type:Thesis of the University of Regensburg (PhD)
Date:2 January 2017
Referee:PD Dr. Oliver J. Bosch
Date of exam:8 April 2016
Institutions:Biology, Preclinical Medicine > Institut für Zoologie > Tierphysiologie/Neurobiologie (Prof. Dr. Inga Neumann)
Projects:Deutsche Forschungsgemeinschaft (DFG BO1958/6-1 to OJB)
Interdisciplinary Subject Network:Immobilien- und Kapitalmärkte
Keywords:BNST;EPM;maternal aggression;maternal behaviour;maternal care;maternal motivation;MPOA;pup retrieval;PVN;vasopressin;V1a receptors;V1b receptors
Dewey Decimal Classification:500 Science > 500 Natural sciences & mathematics
500 Science > 590 Zoological sciences
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:33778
Owner only: item control page

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