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Complement Regulator FHR-3 Is Elevated either Locally or Systemically in a Selection of Autoimmune Diseases

Schmid, Peter M., Bouazzaoui, Karin, Schmid, Karin, Birner, Christoph, Schach, Christian, Maier, Lars S., Holler, Ernst and Endemann, Dierk H. (2017) Complement Regulator FHR-3 Is Elevated either Locally or Systemically in a Selection of Autoimmune Diseases. Cell Transplantation 26 (8), pp. 1428-1440.

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Date of publication of this fulltext: 25 Jan 2018 13:32

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Other URL: http://journals.sagepub.com/doi/abs/10.1177/0963689717720295?url_ver=Z39.88-2003&rfr_id=ori%3Arid%3Acrossref.org&rfr_dat=cr_pub%3Dpubmed&

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Abstract

Acute kidney injury (AKI) is a very common complication after allogeneic bone marrow transplantation (BMT) and is associated with a poor prognosis. Generally, the kidneys are assumed to not be no direct targets of graft-versus-host disease (GvHD), and renal impairment is often attributed to several other factors occurring in the early phase after BMT. Our study aimed to prove the existence of ...

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Item type:Article
Date:13 September 2017
Institutions:Medicine > Lehrstuhl für Innere Medizin II
Projects:Open Access Publizieren (DFG)
Identification Number:
ValueType
10.1177/0963689717720295DOI
Keywords:STEM-CELL TRANSPLANTATION; ACUTE KIDNEY INJURY; REGULATORY T-CELLS; INDOLEAMINE 2,3-DIOXYGENASE GENE; IFN-GAMMA; NEPHROTIC SYNDROME; INTERFERON-GAMMA; SERUM-LEVELS; EXPRESSION; GVHD; renal GvHD; cytokines; IDO; apoptosis; urinary NAG
Dewey Decimal Classification:600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:35154
Owner only: item control page

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