![]() | License: Creative Commons Attribution 4.0 (5MB) |
- URN to cite this document:
- urn:nbn:de:bvb:355-epub-378013
- DOI to cite this document:
- 10.5283/epub.37801
Abstract
Differentiated neurons established via iPSCs from patients that suffer from familial Parkinson's disease (PD) have allowed insights into the mechanisms of neurodegeneration. In the larger cohort of patients with sporadic PD, iPSC based information on disease specific cellular phenotypes is rare. We asked whether differences may be present on genomic and epigenomic levels and performed a ...

Owner only: item control page