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Death receptor 3 mediates necroptotic cell death

Bittner, Sebastian, Knoll, Gertrud and Ehrenschwender, Martin (2017) Death receptor 3 mediates necroptotic cell death. Cellular and Molecular Life Sciences 74 (3), pp. 543-554.

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Other URL: http://doi.org/10.1007/s00018-016-2355-2


Abstract

Death receptor 3 (DR3) was initially identified as a T cell co-stimulatory and pro-inflammatory molecule, but further studies revealed a more complex role of DR3 and its ligand TL1A. Although being a death receptor, DR3 gained to date predominantly attention as a contributor to inflammation-driven diseases. In our study, we investigated the cell death pathways associated with DR3. We show that in ...

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Item type:Article
Date:2017
Institutions:Medicine > Lehrstuhl für Medizinische Mikrobiologie und Hygiene
Identification Number:
ValueType
10.1007/s00018-016-2355-2DOI
Keywords:NF-KAPPA-B; TUMOR-NECROSIS-FACTOR; MIXED LINEAGE KINASE; TNF-INDUCED NECROSIS; PROGRAMMED NECROSIS; REGULATED NECROSIS; DOMAIN-LIKE; ACTIVATION; APOPTOSIS; ALPHA; Death receptor 3; Necroptosis; TL1A; RIP3; MLKL; TNFRSF25
Dewey Decimal Classification:600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:38775
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