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Catecholamine-Dependent β-Adrenergic Signaling in a Pluripotent Stem Cell Model of Takotsubo Cardiomyopathy

Borchert, Thomas, Hübscher, Daniela, Guessoum, Celina I., Lam, Tuan-Dinh D., Ghadri, Jelena R., Schellinger, Isabel N., Tiburcy, Malte, Liaw, Norman Y., Li, Yun, Haas, Jan, Sossalla, Samuel, Huber, Mia A., Cyganek, Lukas, Jacobshagen, Claudius, Dressel, Ralf, Raaz, Uwe, Nikolaev, Viacheslav O., Guan, Kaomei , Thiele, Holger, Meder, Benjamin, Wollnik, Bernd, Zimmermann, Wolfram-Hubertus, Lüscher, Thomas F., Hasenfuss, Gerd, Templin, Christian and Streckfuss-Bömeke, Katrin (2017) Catecholamine-Dependent β-Adrenergic Signaling in a Pluripotent Stem Cell Model of Takotsubo Cardiomyopathy. Journal of the American College of Cardiology 70 (8), pp. 975-991.

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Other URL: http://doi.org/10.1016/j.jacc.2017.06.061


Abstract

BACKGROUND Takotsubo syndrome (TTS) is characterized by an acute left ventricular dysfunction and is associated with life-threating complications in the acute phase. The underlying disease mechanism in TTS is still unknown. A genetic basis has been suggested to be involved in the pathogenesis. OBJECTIVES The aims of the study were to establish an in vitro induced pluripotent stem cell (iPSC) ...

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Item type:Article
Date:2017
Institutions:Medicine > Lehrstuhl für Innere Medizin II
Identification Number:
ValueType
10.1016/j.jacc.2017.06.061DOI
Keywords:ACUTE MYOCARDIAL-INFARCTION; STRESS CARDIOMYOPATHY; MOUSE MODEL; TAKO-TSUBO; CARDIOMYOCYTES; ISOPROTERENOL; PATHOGENESIS; DEFICIENCY; PATHWAY; broken heart syndrome; catecholamine; electrical activity; iPSC cardiomyocytes; lipotoxicity; TTS pathogenesis
Dewey Decimal Classification:600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:39259
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