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Increased cFLIP expression in thymic epithelial tumors blocks autophagy via NF-κB signalling

Belharazem, Djeda, Grass, Albert, Paul, Cornelia, Vitacolonna, Mario, Schalke, Berthold, Rieker, Ralf J., Körner, Daniel, Jungebluth, Philipp, Simon-Keller, Katja , Hohenberger, Peter, Roessner, Eric M., Wiebe, Karsten, Gräter, Thomas, Kyriss, Thomas, Ott, German, Geserick, Peter, Leverkus, Martin, Ströbel, Philipp and Marx, Alexander (2017) Increased cFLIP expression in thymic epithelial tumors blocks autophagy via NF-κB signalling. Oncotarget 8 (52).

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Other URL: http://doi.org/10.18632/oncotarget.15929


Abstract

The anti-apoptotic cellular FLICE-like inhibitory protein cFLIP plays a pivotal role in normal tissues homoeostasis and the development of many tumors, but its role in normal thymus (NT), thymomas and thymic carcinomas (TC) is largely unknown. Expression, regulation and function of cFLIP were analyzed in biopsies of NT, thymomas, thymic squamous cell carcinomas (TSCC), thymic epithelial cells ...

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Item type:Article
Date:2017
Institutions:Medicine > Lehrstuhl für Neurologie
Identification Number:
ValueType
10.18632/oncotarget.15929DOI
Keywords:TRAIL-INDUCED APOPTOSIS; C-FLIP; CANCER-CELLS; CARCINOMA; THYMOMA; RESISTANCE; DEATH; NECROSIS; PATHWAY; PATHOGENESIS; thymic tumors; cFLIP; NF-kappa B; senescence; autophagy
Dewey Decimal Classification:600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:39652
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