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Proline‐dependent and basophilic kinases phosphorylate human TRPC6 at serine 14 to control channel activity through increased membrane expression

Hagmann, Henning ; Mangold, Nicole ; Rinschen, Markus M. ; Koenig, Tim ; Kunzelmann, Karl ; Schermer, Bernhard ; Benzing, Thomas ; Brinkkoetter, Paul T.



Zusammenfassung

Signaling via the transient receptor potential (TRP) ion channel C6 plays a pivotal role in hereditary and sporadic glomerular kidney disease. Several studies have identified gain-of-function mutations of TRPC6 and report induced expression and enhanced channel activity of TRPC6 in association with glomerular diseases. Interfering with TRPC6 activity may open novel therapeutic pathways. TRPC6 ...

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