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HCV-Induced Epigenetic Changes Associated With Liver Cancer Risk Persist After Sustained Virologic Response

Hamdane, Nourdine ; Jühling, Frank ; Crouchet, Emilie ; El Saghire, Houssein ; Thumann, Christine ; Oudot, Marine A. ; Bandiera, Simonetta ; Saviano, Antonio ; Ponsolles, Clara ; Roca Suarez, Armando Andres ; Li, Shen ; Fujiwara, Naoto ; Ono, Atsushi ; Davidson, Irwin ; Bardeesy, Nabeel ; Schmidl, Christian ; Bock, Christoph ; Schuster, Catherine ; Lupberger, Joachim ; Habersetzer, François ; Doffoël, Michel ; Piardi, Tullio ; Sommacale, Daniele ; Imamura, Michio ; Uchida, Takuro ; Ohdan, Hideki ; Aikata, Hiroshi ; Chayama, Kazuaki ; Boldanova, Tujana ; Pessaux, Patrick ; Fuchs, Bryan C. ; Hoshida, Yujin ; Zeisel, Mirjam B. ; Duong, François H.T. ; Baumert, Thomas F.



Abstract

BACKGROUND & AIMS: Chronic hepatitis C virus (HCV) infection is an important risk factor for hepatocellular carcinoma (HCC). Despite effective antiviral therapies, the risk for HCC is decreased but not eliminated after a sustained virologic response (SVR) to direct-acting antiviral (DAA) agents, and the risk is higher in patients with advanced fibrosis. We investigated HCV-induced epigenetic ...

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