Taniyama, Yoshiaki ; Cremer, Sebastian ; Pilgram, Lisa ; Berkowitsch, Alexander ; Stecher, Melanie ; Rieg, Siegbert ; Shumliakivska, Mariana ; Bojkova, Denisa 
; Wagner, Julian Uwe Gabriel ; Aslan, Galip Servet ; Spinner, Christoph ; Luxán, Guillermo ; Hanses, Frank ; Dolff, Sebastian ; Piepel, Christiane ; Ruppert, Clemens ; Guenther, Andreas ; Rüthrich, Maria Madeleine ; Vehreschild, Jörg Janne ; Wille, Kai ; Haselberger, Martina ; Heuzeroth, Hanno ; Hansen, Arne ; Eschenhagen, Thomas ; Cinatl, Jindrich ; Ciesek, Sandra ; Dimmeler, Stefanie ; Borgmann, Stefan ; Zeiher, Andreas
; Wagner, Julian Uwe Gabriel ; Aslan, Galip Servet ; Spinner, Christoph ; Luxán, Guillermo ; Hanses, Frank ; Dolff, Sebastian ; Piepel, Christiane ; Ruppert, Clemens ; Guenther, Andreas ; Rüthrich, Maria Madeleine ; Vehreschild, Jörg Janne ; Wille, Kai ; Haselberger, Martina ; Heuzeroth, Hanno ; Hansen, Arne ; Eschenhagen, Thomas ; Cinatl, Jindrich ; Ciesek, Sandra ; Dimmeler, Stefanie ; Borgmann, Stefan ; Zeiher, Andreas 
Zusammenfassung
Aims Patients with cardiovascular comorbidities have a significantly increased risk for a critical course of COVID-19. As the SARS-CoV2 virus enters cells via the angiotensin-converting enzyme receptor II (ACE2), drugs which interact with the renin angiotensin aldosterone system (RAAS) were suspected to influence disease severity. Methods and results We analyzed 1946 consecutive patients with ...
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