Bischof, Corinne 
; Mirtschink, Peter ; Yuan, Ting ; Wu, Meiqian ; Zhu, Chaonan ; Kaur, Jaskiran ; Pham, Minh Duc ; Gonzalez-Gonoggia, Suam ; Hammer, Marie ; Rogg, Eva-Maria ; Sharma, Rahul ; Bottermann, Katharina ; Gercken, Bettina ; Hagag, Eman ; Berthonneche, Corinne ; Sossalla, Samuel ; Stehr, Sebastian N. ; Maxeiner, Joachim ; Duda, Maria Anna ; Latreille, Mathieu ; Zamboni, Nicola ; Martelli, Fabio ; Pedrazzini, Thierry ; Dimmeler, Stefanie ; Krishnan, Jaya
; Mirtschink, Peter ; Yuan, Ting ; Wu, Meiqian ; Zhu, Chaonan ; Kaur, Jaskiran ; Pham, Minh Duc ; Gonzalez-Gonoggia, Suam ; Hammer, Marie ; Rogg, Eva-Maria ; Sharma, Rahul ; Bottermann, Katharina ; Gercken, Bettina ; Hagag, Eman ; Berthonneche, Corinne ; Sossalla, Samuel ; Stehr, Sebastian N. ; Maxeiner, Joachim ; Duda, Maria Anna ; Latreille, Mathieu ; Zamboni, Nicola ; Martelli, Fabio ; Pedrazzini, Thierry ; Dimmeler, Stefanie ; Krishnan, Jaya 
Abstract
Endoreplication, duplication of the nuclear genome without cell division, occurs in disease to drive morphologic growth, cell fate, and function. Despite its criticality, the metabolic underpinnings of disease-induced endoreplication and its link to morphologic growth are unknown. Heart disease is characterized by endoreplication preceding cardiac hypertrophy. We identify ATP synthase as a ...
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