Kisand, Kai 
; Bøe Wolff, Anette S. ; Podkrajšek, Katarina Trebušak ; Tserel, Liina ; Link, Maire ; Kisand, Kalle V. ; Ersvaer, Elisabeth ; Perheentupa, Jaakko ; Erichsen, Martina Moter ; Bratanic, Nina ; Meloni, Antonella ; Cetani, Filomena ; Perniola, Roberto ; Ergun-Longmire, Berrin ; Maclaren, Noel ; Krohn, Kai J. E. ; Pura, Mikuláš ; Schalke, Berthold ; Ströbel, Philipp ; Leite, Maria Isabel ; Battelino, Tadej ; Husebye, Eystein S. ; Peterson, Pärt ; Willcox, Nick ; Meager, Anthony
; Bøe Wolff, Anette S. ; Podkrajšek, Katarina Trebušak ; Tserel, Liina ; Link, Maire ; Kisand, Kalle V. ; Ersvaer, Elisabeth ; Perheentupa, Jaakko ; Erichsen, Martina Moter ; Bratanic, Nina ; Meloni, Antonella ; Cetani, Filomena ; Perniola, Roberto ; Ergun-Longmire, Berrin ; Maclaren, Noel ; Krohn, Kai J. E. ; Pura, Mikuláš ; Schalke, Berthold ; Ströbel, Philipp ; Leite, Maria Isabel ; Battelino, Tadej ; Husebye, Eystein S. ; Peterson, Pärt ; Willcox, Nick ; Meager, Anthony 
Abstract
Chronic mucocutaneous candidiasis (CMC) is frequently associated with T cell immunodeficiencies. Specifically, the proinflammatory IL-17A-producing Th17 subset is implicated in protection against fungi at epithelial surfaces. In autoimmune polyendocrinopathy candidiasis ectodermal dystrophy (APECED, or autoimmune polyendocrine syndrome 1), CMC is often the first sign, but the underlying ...
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