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Höcherl, Klaus ; Schmidt, Christoph ; Kurt, Birgül ; Bucher, Michael

Activation of the PGI 2 /IP System Contributes to the Development of Circulatory Failure in a Rat Model of Endotoxic Shock

Höcherl, Klaus, Schmidt, Christoph, Kurt, Birgül and Bucher, Michael (2008) Activation of the PGI 2 /IP System Contributes to the Development of Circulatory Failure in a Rat Model of Endotoxic Shock. Hypertension 52 (2), pp. 330-335.

Date of publication of this fulltext: 19 Dec 2024 13:14
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Item typeArticle
Journal or Publication TitleHypertension
Publisher:LIPPINCOTT WILLIAMS & WILKINS
Place of Publication:PHILADELPHIA
Volume:52
Number of Issue or Book Chapter:2
Page Range:pp. 330-335
Date2008
InstitutionsMedicine > Lehrstuhl für Anästhesiologie
Biology, Preclinical Medicine > Institut für Physiologie
Identification Number
ValueType
10.1161/HYPERTENSIONAHA.108.112029DOI
KeywordsNITRIC-OXIDE SYNTHASE; NECROSIS-FACTOR-ALPHA; SMOOTH-MUSCLE-CELLS; PROSTACYCLIN SYNTHASE; ENDOTHELIAL-CELLS; CYCLOOXYGENASE INHIBITION; PROSTAGLANDIN I-2; SEPTIC SHOCK; RECEPTOR; SEPSIS; prostacyclin; LPS; CAY-10441; blood pressure; sepsis
Dewey Decimal Classification500 Science > 570 Life sciences
600 Technology > 610 Medical sciences Medicine
StatusPublished
RefereedYes, this version has been refereed
Created at the University of RegensburgYes
Item ID67948

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