Publikationen von 0000-0003-0728-8361
(ORCID: 0000-0003-0728-8361)
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Anzahl der Einträge: 3.
2017
Che, Li, Pilo, Maria G., Cigliano, Antonio
, Latte, Gavinella
, Simile, Maria M., Ribback, Silvia, Dombrowski, Frank, Evert, Matthias, Chen, Xin und Calvisi, Diego F.
(2017)
Oncogene dependent requirement of fatty acid synthase in hepatocellular carcinoma.
Cell Cycle 16 (6), S. 499-507.
Volltext nicht vorhanden.
, Latte, Gavinella
, Simile, Maria M., Ribback, Silvia, Dombrowski, Frank, Evert, Matthias, Chen, Xin und Calvisi, Diego F.
(2017)
Oncogene dependent requirement of fatty acid synthase in hepatocellular carcinoma.
Cell Cycle 16 (6), S. 499-507.
Volltext nicht vorhanden.
2016
Wang, Chunmei, Che, Li, Hu, Junjie, Zhang, Shanshan, Jiang, Lijie, Latte, Gavinella
, Demartis, Maria I., Tao, Junyan, Gui, Bing, Pilo, Maria G., Ribback, Silvia, Dombrowski, Frank, Evert, Matthias, Calvisi, Diego F. und Chen, Xin
(2016)
Activated mutant forms of PIK3CA cooperate with RasV12 or c-Met to induce liver tumour formation in mice via AKT2/mTORC1 cascade.
Liver International 36 (8), S. 1176-1186.
Volltext nicht vorhanden.
, Demartis, Maria I., Tao, Junyan, Gui, Bing, Pilo, Maria G., Ribback, Silvia, Dombrowski, Frank, Evert, Matthias, Calvisi, Diego F. und Chen, Xin
(2016)
Activated mutant forms of PIK3CA cooperate with RasV12 or c-Met to induce liver tumour formation in mice via AKT2/mTORC1 cascade.
Liver International 36 (8), S. 1176-1186.
Volltext nicht vorhanden.
Hu, Junjie, Che, Li, Li, Lei, Pilo, Maria G., Cigliano, Antonio
, Ribback, Silvia, Li, Xiaolei, Latte, Gavinella
, Mela, Marta, Evert, Matthias, Dombrowski, Frank, Zheng, Guohua, Chen, Xin und Calvisi, Diego F.
(2016)
Co-activation of AKT and c-Met triggers rapid hepatocellular carcinoma development via the mTORC1/FASN pathway in mice.
Scientific Reports 6 (1).
Volltext nicht vorhanden.
, Ribback, Silvia, Li, Xiaolei, Latte, Gavinella
, Mela, Marta, Evert, Matthias, Dombrowski, Frank, Zheng, Guohua, Chen, Xin und Calvisi, Diego F.
(2016)
Co-activation of AKT and c-Met triggers rapid hepatocellular carcinoma development via the mTORC1/FASN pathway in mice.
Scientific Reports 6 (1).
Volltext nicht vorhanden.
