Einträge von Rick, Ferenc G. auf dem Publikationsserver
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Anzahl der Einträge: 5.
2013
Seitz, Stephan, Buchholz, Stefan, Schally, Andrew V. 
, Jayakumar, Arumugam R., Weber, Florian, Papadia, Andrea , Rick, Ferenc G. , Szalontay, Luca, Treszl, Andrea, Köster, Frank, Ortmann, Olaf und Hohla, Florian
(2013)
Targeting triple-negative breast cancer through the somatostatin receptor with the new cytotoxic somatostatin analogue AN-162 [AEZS-124].
Anti-Cancer Drugs 24 (2), S. 150-157.
Volltext nicht vorhanden.
, Jayakumar, Arumugam R., Weber, Florian, Papadia, Andrea , Rick, Ferenc G. , Szalontay, Luca, Treszl, Andrea, Köster, Frank, Ortmann, Olaf und Hohla, Florian
(2013)
Targeting triple-negative breast cancer through the somatostatin receptor with the new cytotoxic somatostatin analogue AN-162 [AEZS-124].
Anti-Cancer Drugs 24 (2), S. 150-157.
Volltext nicht vorhanden.
2012
Rick, Ferenc G. , Buchholz, Stefan, Schally, Andrew V. , Szalontay, Luca, Krishan, Awtar, Datz, Christian, Stadlmayr, Andreas, Aigner, Elmar, Perez, Roberto, Seitz, Stephan, Block, Norman L. und Hohla, Florian
(2012)
Combination of gastrin-releasing peptide antagonist with cytotoxic agents produces synergistic inhibition of growth of human experimental colon cancers.
Cell Cycle 11 (13), S. 2518-2525.
Volltext nicht vorhanden.
, Buchholz, Stefan, Schally, Andrew V. , Szalontay, Luca, Krishan, Awtar, Datz, Christian, Stadlmayr, Andreas, Aigner, Elmar, Perez, Roberto, Seitz, Stephan, Block, Norman L. und Hohla, Florian
(2012)
Combination of gastrin-releasing peptide antagonist with cytotoxic agents produces synergistic inhibition of growth of human experimental colon cancers.
Cell Cycle 11 (13), S. 2518-2525.
Volltext nicht vorhanden.
Rick, Ferenc G. , Seitz, Stephan, Schally, Andrew V. , Szalontay, Luca, Krishan, Awtar, Datz, Christian, Stadlmayr, Andreas, Buchholz, Stefan, Block, Norman L. und Hohla, Florian
(2012)
GHRH antagonist when combined with cytotoxic agents induces S-phase arrest and additive growth inhibition of human colon cancer.
Cell Cycle 11 (22), S. 4203-4210.
Volltext nicht vorhanden.
, Seitz, Stephan, Schally, Andrew V. , Szalontay, Luca, Krishan, Awtar, Datz, Christian, Stadlmayr, Andreas, Buchholz, Stefan, Block, Norman L. und Hohla, Florian
(2012)
GHRH antagonist when combined with cytotoxic agents induces S-phase arrest and additive growth inhibition of human colon cancer.
Cell Cycle 11 (22), S. 4203-4210.
Volltext nicht vorhanden.
2010
Hohla, Florian, Buchholz, Stefan, Schally, Andrew V. , Krishan, Awtar, Rick, Ferenc G. , Szalontay, Luca, Papadia, Andrea , Halmos, Gabor, Koster, Frank, Aigner, Elmar, Datz, Christian und Seitz, Stephan
(2010)
Targeted cytotoxic somatostatin analog AN-162 inhibits growth of human colon carcinomas and increases sensitivity of doxorubicin resistant murine leukemia cells.
Cancer Letters 294 (1), S. 35-42.
Volltext nicht vorhanden.
, Krishan, Awtar, Rick, Ferenc G. , Szalontay, Luca, Papadia, Andrea , Halmos, Gabor, Koster, Frank, Aigner, Elmar, Datz, Christian und Seitz, Stephan
(2010)
Targeted cytotoxic somatostatin analog AN-162 inhibits growth of human colon carcinomas and increases sensitivity of doxorubicin resistant murine leukemia cells.
Cancer Letters 294 (1), S. 35-42.
Volltext nicht vorhanden.
2009
Hohla, Florian, Buchholz, Stefan, Schally, Andrew V , Seitz, Stefan, Rick, Ferenc G. , Szalontay, Luca, Varga, Jozsef L., Zarandi, Marta, Halmos, Gabor, Vidaurre, Irving, Krishan, Awtar, Kurtoglu, Metin, Chandna, Sudhir , Aigner, Elmar und Datz, Christian
(2009)
GHRH antagonist causes DNA damage leading to p21 mediated cell cycle arrest and apoptosis in human colon cancer cells.
Cell Cycle 8 (19), S. 3149-3156.
Volltext nicht vorhanden.
, Seitz, Stefan, Rick, Ferenc G. , Szalontay, Luca, Varga, Jozsef L., Zarandi, Marta, Halmos, Gabor, Vidaurre, Irving, Krishan, Awtar, Kurtoglu, Metin, Chandna, Sudhir , Aigner, Elmar und Datz, Christian
(2009)
GHRH antagonist causes DNA damage leading to p21 mediated cell cycle arrest and apoptosis in human colon cancer cells.
Cell Cycle 8 (19), S. 3149-3156.
Volltext nicht vorhanden.