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ICAM-1 and VCAM-1 expression following aneurysmal subarachnoid hemorrhage and their possible role in the pathophysiology of subsequent ischemic deficits

DOI to cite this document:
10.5283/epub.1235
Rothoerl, Ralf Dirk ; Schebesch, Karl-Michael ; Kubitza, Marion ; Woertgen, Chris ; Brawanski, Alexander ; Pina, Ana-Luisa
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Date of publication of this fulltext: 05 Aug 2009 13:26


Abstract

BACKGROUND: The pathophysiology of ischemic cerebral lesions following aneurysmal subarachnoid hemorrhage (SAH) is poorly understood. There is growing evidence that inflammatory reactions could be involved in the pathogenesis of such delayed occurring ischemic lesions. The aim of this study was to evaluate adhesion molecules with regard to these lesions following SAH. METHODS: Serum and ...

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