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The podocyte-specific inactivation of Lmx1b, Ldb1 and E2a yields new insight into a transcriptional network in podocytes

Suleiman, Hani ; Heudobler, Daniel ; Raschta, Anne-Sarah ; Zhao, Yangu ; Zhao, Qi ; Hertting, Irmgard ; Vitzthum, Helga ; Moeller, Marcus J. ; Holzman, Lawrence B. ; Rachel, Reinhard ; Johnson, Randy ; Westphal, Heiner ; Rascle, Anne ; Witzgall, Ralph



Abstract

Patients with nail-patella syndrome, which among other symptoms also includes podocyte-associated renal failure, suffer from mutations in the LMX1B gene. The disease severity among patients is quite variable and has given rise to speculations on the presence of modifier genes. Promising candidates for modifier proteins are the proteins interacting with LMX1B, such as LDB1 and E47. Since human ...

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