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Downregulation of RUNX3 and TES by hypermethylation in glioblastoma

Mueller, W., Nutt, C. L., Ehrich, M., Riemenschneider, Markus J., von Deimling, A., van den Boom, D. and Louis, D. N. (2007) Downregulation of RUNX3 and TES by hypermethylation in glioblastoma. Oncogene 26 (4), pp. 583-593.

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Abstract

Glioblastoma, the most aggressive and least treatable form of malignant glioma, is the most common human brain tumor. Although many regions of allelic loss occur in glioblastomas, relatively few tumor suppressor genes have been found mutated at such loci. To address the possibility that epigenetic alterations are an alternative means of glioblastoma gene inactivation, we coupled pharmacological ...

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Item type:Article
Date:2007
Institutions:Medicine > Zentren des Universitätsklinikums Regensburg > Zentrum für Hirntumore (ZHT)
Identification Number:
ValueType
16909125PubMed ID
10.1038/sj.onc.1209805DOI
Classification:
NotationType
Azacitidine/pharmacologyMESH
Brain/metabolismMESH
Core Binding Factor Alpha 3 Subunit/metabolismMESH
Cytoskeletal ProteinsMESH
DNA MethylationMESH
Gene Expression Regulation, NeoplasticMESH
Glioblastoma/pathologyMESH
Homeodomain Proteins/metabolismMESH
HumansMESH
LIM Domain ProteinsMESH
Oligonucleotide Array Sequence AnalysisMESH
Promoter Regions, GeneticMESH
Reverse Transcriptase Polymerase Chain ReactionMESH
Sequence Analysis, DNAMESH
Spectrometry, Mass, Matrix-Assisted Laser Desorption-IonizationMESH
Tumor Cells, CulturedMESH
Tumor Suppressor Proteins/metabolismMESH
Dewey Decimal Classification:600 Technology > 610 Medical sciences Medicine
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Unknown
Item ID:29246
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