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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-308502
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.30850
Zusammenfassung
Background: Heart failure (CHF) is characterized by dyspnea and pulmonary changes. The underlying molecular adaptations are unclear, but might provide targets for therapeutic interventions. We therefore conceived a study to determine molecular changes of early pulmonary stress failure in a model of tachycardia-induced heart failure. Methods: CHF was induced in rabbits by progessive right ...
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