| License: Allianz- bzw. Nationallizenz PDF - Published Version (242kB) |
- URN to cite this document:
- urn:nbn:de:bvb:355-epub-356173
- DOI to cite this document:
- 10.5283/epub.35617
Alternative links to fulltext:DOI
Abstract
Background Myelin oligodendrocyte glycoprotein (MOG) is a candidate primary target of the autoimmune attack on the central nervous system (CNS) in multiple sclerosis (MS). However, the physiological function of MOG has been unclear for a long time. Objective We propose that MOG has a central role in the regulation of tolerance and autoimmunity. Conclusion The interaction of MOG with DC-SIGN, an ...

Owner only: item control page