Hepatic steatosis causes induction of the chemokine RANTES in the absence of significant hepatic inflammation

URN to cite this document:: urn:nbn:de:bvb:355-epub-370480
DOI to cite this document:: 10.5283/epub.37048

Kirovski, Georgi ; Gäbele, Erwin ; Dorn, Christoph

; Moleda, Lukas ; Niessen, Christoph ; Weiss, Thomas S.

; Wobser, Hella ; Schacherer, Doris ; Buechler, Christa

; Wasmuth, Hermann E. ; Hellerbrand, Claus

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Date of publication of this fulltext: 10 Apr 2018 11:50

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Item type:	Article
Journal or Publication Title:	International Journal of Clinical and Experimental Pathology
Publisher:	e-Century Publishing Corporation
Volume:	3
Number of Issue or Book Chapter:	7
Page Range:	pp. 675-680
Date:	2 August 2010
Institutions:	Chemistry and Pharmacy > Institute of Pharmacy > Group Clinical Pharmacy (Dr. Dorn)
Keywords:	Hepatic steatosis, liver cirrhosis, nonalcoholic steatosis, steatohepatitis, chemokine CCL5, RANTES, inflammation, Nonalcoholic fatty liver disease (NAFLD)
Dewey Decimal Classification:	600 Technology > 615 Pharmacy
Status:	Published
Refereed:	Yes, this version has been refereed
Created at the University of Regensburg:	Yes
Item ID:	37048

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Abstract

Nonalcoholic fatty liver disease (NAFLD) encompasses a spectrum ranging from simple steatosis to cirrhosis. Hepatocellular lipid accumulation is a hallmark of both nonalcoholic steatosis and steatohepatitis (NASH). The latter develops upon pro-inflammatory cell infiltration and is widely considered as the first relevant pathophysiological step in NAFLD-progression. The chemokine CCL5/RANTES plays ...

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