| Lizenz: Creative Commons Namensnennung 4.0 International (518kB) |
- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-379332
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.37933
Zusammenfassung
Aims In hypertrophy and heart failure, the proarrhythmic persistent Na+ current (I-NaL) is enhanced. We aimed to investigate the electrophysiological role of neuronal sodium channel Na(V)1.8 in human hypertrophied myocardium. Methods and results Myocardial tissue of 24 patients suffering from symptomatic severe aortic stenosis and concomitant significant afterload-induced hypertrophy with ...
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