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The functional consequences of sodium channel NaV 1.8 in human left ventricular hypertrophy

URN to cite this document:
urn:nbn:de:bvb:355-epub-379332
DOI to cite this document:
10.5283/epub.37933
Ahmad, Shakil ; Tirilomis, Petros ; Pabel, Steffen ; Dybkova, Nataliya ; Hartmann, Nico ; Molina, Cristina E. ; Tirilomis, Theodoros ; Kutschka, Ingo ; Frey, Norbert ; Maier, Lars S. ; Hasenfuss, Gerd ; Streckfuss-Bömeke, Katrin ; Sossalla, Samuel
Date of publication of this fulltext: 07 Nov 2018 13:39



Abstract

Aims In hypertrophy and heart failure, the proarrhythmic persistent Na+ current (I-NaL) is enhanced. We aimed to investigate the electrophysiological role of neuronal sodium channel Na(V)1.8 in human hypertrophied myocardium. Methods and results Myocardial tissue of 24 patients suffering from symptomatic severe aortic stenosis and concomitant significant afterload-induced hypertrophy with ...

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