Go to content
UR Home

Salt-responsive gut commensal modulates TH17 axis and disease

Wilck, Nicola, Matus, Mariana G., Kearney, Sean M., Olesen, Scott W. , Forslund, Kristoffer, Bartolomaeus, Hendrik , Haase, Stefanie, Mähler, Anja, Balogh, András , Markó, Lajos , Vvedenskaya, Olga, Kleiner, Friedrich H., Tsvetkov, Dmitry, Klug, Lars, Costea, Paul I., Sunagawa, Shinichi , Maier, Lisa, Rakova, Natalia, Schatz, Valentin, Neubert, Patrick, Frätzer, Christian, Krannich, Alexander, Gollasch, Maik, Grohme, Diana A., Côrte-Real, Beatriz F., Gerlach, Roman G., Basic, Marijana, Typas, Athanasios, Wu, Chuan, Titze, Jens M., Jantsch, Jonathan, Boschmann, Michael, Dechend, Ralf, Kleinewietfeld, Markus , Kempa, Stefan, Bork, Peer, Linker, Ralf A., Alm, Eric J. and Müller, Dominik N. (2017) Salt-responsive gut commensal modulates TH17 axis and disease. Nature 551 (7682), pp. 585-589.

Full text not available from this repository.

at publisher (via DOI)

Other URL: http://doi.org/10.1038/nature24628


Abstract

A Western lifestyle with high salt consumption can lead to hypertension and cardiovascular disease. High salt may additionally drive autoimmunity by inducing T helper 17 (T(H)17) cells, which can also contribute to hypertension. Induction of T(H)17 cells depends on gut microbiota; however, the effect of salt on the gut microbiome is unknown. Here we show that high salt intake affects the gut ...

plus


Export bibliographical data



Item type:Article
Date:2017
Institutions:Medicine > Lehrstuhl für Medizinische Mikrobiologie und Hygiene
Identification Number:
ValueType
10.1038/nature24628DOI
Keywords:II-INDUCED HYPERTENSION; ARYL-HYDROCARBON RECEPTOR; BLOOD-PRESSURE; MULTIPLE-SCLEROSIS; VASCULAR DYSFUNCTION; SODIUM-INTAKE; MARKER GENES; T-CELLS; MICROBIOTA; LACTOBACILLI;
Dewey Decimal Classification:500 Science > 570 Life sciences
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:39553
Owner only: item control page
  1. Homepage UR

University Library

Publication Server

Contact:

Publishing: oa@ur.de

Dissertations: dissertationen@ur.de

Research data: daten@ur.de

Contact persons