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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-415013
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.41501
Zusammenfassung
There is recent evidence that the dysfunctional responses of a peculiar visceral fat deposit known as epicardial adipose tissue (EAT) can directly promote cardiac enlargement in the case of obesity. Here, we observed a newer molecular pattern associated with LV dysfunction mediated by prostaglandin E2 (PGE(2)) deregulation in EAT in a cardiovascular disease (CVD) population. A series of 33 ...
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