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- URN to cite this document:
- urn:nbn:de:bvb:355-epub-448037
- DOI to cite this document:
- 10.5283/epub.44803
This publication is part of the DEAL contract with Springer.
Abstract
Pharmacologic approaches for the treatment of atrial arrhythmias are limited due to side effects and low efficacy. Thus, the identification of new antiarrhythmic targets is of clinical interest. Recent genome studies suggested an involvement of SCN10A sodium channels (Na(V)1.8) in atrial electrophysiology. This study investigated the role and involvement of Na(V)1.8 (SCN10A) in arrhythmia ...
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