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Selective Activation of Tumor Necrosis Factor Receptor II Induces Antiinflammatory Responses and Alleviates Experimental Arthritis

Fischer, Roman ; Proske, Marcel ; Duffey, Maëlle ; Stangl, Hubert ; Martinez, George F. ; Peters, Nathalie ; Kraske, Alexandra ; Straub, Rainer H. ; Bethea, John R. ; Kontermann, Roland E. ; Pfizenmaier, Klaus


Objective. Treg cells modulate immune responses and can suppress the development of autoimmune diseases. Tumor necrosis factor receptor II (TNFRII) has been recognized as a key receptor on these cells that facilitates expansion and stabilization of CD4+ Treg cells. The purpose of the present study was to investigate the therapeutic activity of a novel TNFRII agonist in experimental arthritis as ...


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