Mayén, Ana‐Lucia 
; Aglago, Elom K. ; Knaze, Viktoria ; Cordova, Reynalda ; Schalkwijk, Casper G. ; Wagner, Karl‐Heinz ; Aleksandrova, Krasimira ; Fedirko, Veronika ; Keski‐Rahkonen, Pekka ; Leitzmann, Michael F. ; Katzke, Verena ; Srour, Bernard ; Schulze, Matthias B. ; Masala, Giovanna ; Krogh, Vittorio ; Panico, Salvatore ; Tumino, Rosario ; Bueno‐de‐Mesquita, Bas ; Brustad, Magritt ; Agudo, Antonio ; Chirlaque López, María Dolores ; Amiano, Pilar ; Ohlsson, Bodil ; Ramne, Stina ; Aune, Dagfinn ; Weiderpass, Elisabete ; Jenab, Mazda ; Freisling, Heinz
; Aglago, Elom K. ; Knaze, Viktoria ; Cordova, Reynalda ; Schalkwijk, Casper G. ; Wagner, Karl‐Heinz ; Aleksandrova, Krasimira ; Fedirko, Veronika ; Keski‐Rahkonen, Pekka ; Leitzmann, Michael F. ; Katzke, Verena ; Srour, Bernard ; Schulze, Matthias B. ; Masala, Giovanna ; Krogh, Vittorio ; Panico, Salvatore ; Tumino, Rosario ; Bueno‐de‐Mesquita, Bas ; Brustad, Magritt ; Agudo, Antonio ; Chirlaque López, María Dolores ; Amiano, Pilar ; Ohlsson, Bodil ; Ramne, Stina ; Aune, Dagfinn ; Weiderpass, Elisabete ; Jenab, Mazda ; Freisling, Heinz 
Zusammenfassung
Advanced glycation endproducts (AGEs) may contribute to liver carcinogenesis because of their proinflammatory and prooxidative properties. Diet is a major source of AGEs, but there is sparse human evidence on the role of AGEs intake in liver cancer etiology. We examined the association between dietary AGEs and the risk of hepatobiliary cancers in the European Prospective Investigation into Cancer ...
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