Hennig, Thomas ; Prusty, Archana B. 
; Kaufer, Benedikt B. ; Whisnant, Adam W. ; Lodha, Manivel ; Enders, Antje ; Thomas, Julius ; Kasimir, Francesca ; Grothey, Arnhild ; Klein, Teresa ; Herb, Stefanie ; Jürges, Christopher ; Sauer, Markus ; Fischer, Utz ; Rudel, Thomas ; Meister, Gunter ; Erhard, Florian ; Dölken, Lars ; Prusty, Bhupesh K.
; Kaufer, Benedikt B. ; Whisnant, Adam W. ; Lodha, Manivel ; Enders, Antje ; Thomas, Julius ; Kasimir, Francesca ; Grothey, Arnhild ; Klein, Teresa ; Herb, Stefanie ; Jürges, Christopher ; Sauer, Markus ; Fischer, Utz ; Rudel, Thomas ; Meister, Gunter ; Erhard, Florian ; Dölken, Lars ; Prusty, Bhupesh K. 
Zusammenfassung
Herpesviruses have mastered host cell modulation and immune evasion to augment productive infection, life-long latency and reactivation(1,2). A long appreciated, yet undefined relationship exists between the lytic-latent switch and viral non-coding RNAs3,4. Here we identify viral microRNA (miRNA)-mediated inhibition of host miRNA processing as a cellular mechanism that human herpesvirus 6A ...
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