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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-589126
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.58912
Zusammenfassung
Abstract Background Tumor necrosis factor alpha (TNF) is able to kill cancer cells via receptor-mediated cell death requiring adenosine triphosphate (ATP). Clinical usage of TNF so far is largely limited by its profound hepatotoxicity. Recently, it was found in the murine system that specific protection of hepatocytes against TNF's detrimental effects can be achieved by fructose-mediated ATP ...
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