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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-589839
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.58983
Zusammenfassung
Abstract Amyloid beta (Aβ) plays a major role in the pathogenesis of Alzheimer’s disease and, more recently, has been shown to protect against liver fibrosis. Therefore, we studied Aβ-42 levels and the expression of genes involved in the generation, degradation, and transport of Aβ proteins in liver samples from patients at different stages of metabolic dysfunction-associated liver disease ...
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