Binger, Katrina J. 
; Gebhardt, Matthias ; Heinig, Matthias ; Rintisch, Carola ; Schroeder, Agnes ; Neuhofer, Wolfgang ; Hilgers, Karl ; Manzel, Arndt ; Schwartz, Christian ; Kleinewietfeld, Markus ; Voelkl, Jakob ; Schatz, Valentin ; Linker, Ralf A. ; Lang, Florian ; Voehringer, David ; Wright, Mark D. ; Hubner, Norbert ; Dechend, Ralf ; Jantsch, Jonathan ; Titze, Jens ; Müller, Dominik N.
; Gebhardt, Matthias ; Heinig, Matthias ; Rintisch, Carola ; Schroeder, Agnes ; Neuhofer, Wolfgang ; Hilgers, Karl ; Manzel, Arndt ; Schwartz, Christian ; Kleinewietfeld, Markus ; Voelkl, Jakob ; Schatz, Valentin ; Linker, Ralf A. ; Lang, Florian ; Voehringer, David ; Wright, Mark D. ; Hubner, Norbert ; Dechend, Ralf ; Jantsch, Jonathan ; Titze, Jens ; Müller, Dominik N. 
Zusammenfassung
A high intake of dietary salt (NaCl) has been implicated in the development of hypertension, chronic inflammation, and autoimmune diseases. We have recently shown that salt has a proinflammatory effect and boosts the activation of Th17 cells and the activation of classical, LPS-induced macrophages (M1). Here, we examined how the activation of alternative (M2) macrophages is affected by salt. In ...
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