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Inhibition of TEGDMA and HEMA-induced genotoxicity and cell cycle arrest by N-acetylcysteine

Schweikl, H. ; Hartmann, A. ; Hiller, K.-A. ; Spagnuolo, G. ; Bolay, C. ; Brockhoff, G. ; Schmalz, G.



Zusammenfassung

Objectives. Dental resin monomers like triethylene glycol dimethacrylate (TEGDMA) and 2-hydroxyethyl methacrylate (HEMA) are able to cause an imbalance of the redox state in mammalian cells. The resulting oxidative stress originating from reactive oxygen species (ROS) has been associated with cytotoxicity. We hypothesized that ROS might contribute to the generation of genotoxicity by TEGDMA and ...

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