Paust, Hans-Joachim ; Song, Ning ; De Feo, Donatella ; Asada, Nariaki 
; Tuzlak, Selma ; Zhao, Yu ; Riedel, Jan-Hendrik ; Hellmig, Malte ; Sivayoganathan, Amirrtavarshni ; Peters, Anett ; Kaffke, Anna ; Borchers, Alina ; Wenzel, Ulrich O. ; Steinmetz, Oliver M. ; Tiegs, Gisa ; Meister, Elisabeth ; Mack, Matthias ; Kurts, Christian ; von Vietinghoff, Sibylle ; Lindenmeyer, Maja T. ; Hoxha, Elion ; Stahl, Rolf A. K. ; Huber, Tobias B. ; Bonn, Stefan ; Meyer-Schwesinger, Catherine ; Wiech, Thorsten ; Turner, Jan-Eric ; Becher, Burkhard ; Krebs, Christian F. ; Panzer, Ulf
; Tuzlak, Selma ; Zhao, Yu ; Riedel, Jan-Hendrik ; Hellmig, Malte ; Sivayoganathan, Amirrtavarshni ; Peters, Anett ; Kaffke, Anna ; Borchers, Alina ; Wenzel, Ulrich O. ; Steinmetz, Oliver M. ; Tiegs, Gisa ; Meister, Elisabeth ; Mack, Matthias ; Kurts, Christian ; von Vietinghoff, Sibylle ; Lindenmeyer, Maja T. ; Hoxha, Elion ; Stahl, Rolf A. K. ; Huber, Tobias B. ; Bonn, Stefan ; Meyer-Schwesinger, Catherine ; Wiech, Thorsten ; Turner, Jan-Eric ; Becher, Burkhard ; Krebs, Christian F. ; Panzer, Ulf 
Zusammenfassung
Glomerulonephritis is a group of immune-mediated diseases that cause inflammation within the glomerulus and adjacent compartments of the kidney and is a major cause of end-stage renal disease. T cells are among the main drivers of glomerulonephritis. However, the T cell subsets, cytokine networks, and downstream effector mechanisms that lead to renal tissue injury are largely unknown, which has ...
Nur für Besitzer und Autoren: Kontrollseite des Eintrags
Altmetric