| Lizenz: Creative Commons Namensnennung 4.0 International PDF - Veröffentlichte Version (313kB) |
- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-766075
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.76607
Zusammenfassung
Aim: An excessively activated or dysregulated complement system has been proven to be a vital contributor to the pathogenesis of periodontitis. It has been previously hypothesized that inhibiting the activity of complement component C5 by targeting the C5a receptor is a powerful candidate for treating periodontitis. Here, we apply the drug target instrumental variable (IV) approach to investigate ...

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