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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-789308
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.78930
Zusammenfassung
Cancer cells rely on citrate for multiple metabolic processes, suggesting that limiting the availability of extracellular citrate may represent a novel therapeutic strategy. The plasma membrane citrate transporter (pmCiC) has been implicated in the pathogenesis of several cancers before, and its activity can be inhibited by gluconate. Tissue samples from patients were stained, and pmCiC ...
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