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Structural magnetic resonance imaging in amyotrophic lateral sclerosis: cortical morphometry, diffusion properties and lesion detection as potential biomarkers for the state and progression of amyotrophic lateral sclerosis

Wirth, Anna Maria (2019) Structural magnetic resonance imaging in amyotrophic lateral sclerosis: cortical morphometry, diffusion properties and lesion detection as potential biomarkers for the state and progression of amyotrophic lateral sclerosis. PhD, Universität Regensburg.

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Date of publication of this fulltext: 27 May 2019 12:20

Abstract (English)

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disease primarily affecting the motor system of the central nervous system. The aim of this thesis is to evaluate the potential of structural magnetic resonance imaging (MRI) biomarkers of the brain grey matter (GM) and white matter (WM) for the state and progression of ALS. The thesis has been conducted on behalf of a ...

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Translation of the abstract (German)

Amyotrophe Lateralsklerose (ALS) ist eine progressive neurodegenerative Erkrankung, die primär das motorische System des zentralen Nervensystems betrifft. Ziel dieser Abschlussarbeit ist die Evaluation des Potenzials von Biomarkern der grauen und weißen Substanz für das Stadium und den Verlauf der Amyotrophen Lateralsklerose mittels der strukturellen Magnetresonanztomographie (MRT). Die ...

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Item type:Thesis of the University of Regensburg (PhD)
Date:27 May 2019
Referee:Prof. Dr. Mark W. Greenlee
Date of exam:13 May 2019
Institutions:Psychology and Pedagogy > Institut für Psychologie
Psychology and Pedagogy > Institut für Psychologie > Lehrstuhl für Psychologie I (Allgemeine Psychologie I und Methodenlehre) - Prof. Dr. Mark W. Greenlee
Keywords:Amyotrophic Lateral Sclerosis, Magnetic Resonance Imaging, Biomarker
Dewey Decimal Classification:100 Philosophy & psychology > 150 Psychology
Status:Published
Refereed:Yes, this version has been refereed
Created at the University of Regensburg:Yes
Item ID:40215
Owner only: item control page

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