| License: Creative Commons Attribution 4.0 PDF - Published Version (9MB) |
- URN to cite this document:
- urn:nbn:de:bvb:355-epub-415936
- DOI to cite this document:
- 10.5283/epub.41593
Abstract
Oxidative stress-induced damage of the retinal pigment epithelium (RPE) and chronic inflammation have been suggested as major contributors to a range of retinal diseases. Here, we examined the effects of oxidative stress on endogenous complement components and proinflammatory and angiogenic responses in RPE cells. ARPE-19 cells exposed for 1-48 h to H2O2 had reduced cell-cell contact and ...
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