Petersen, Ann-Kristin ; Stark, Klaus ; Musameh, Muntaser D. ; Nelson, Christopher P. 
; Römisch-Margl, Werner ; Kremer, Werner ; Raffler, Johannes ; Krug, Susanne ; Skurk, Thomas ; Rist, Manuela J. ; Daniel, Hannelore ; Hauner, Hans ; Adamski, Jerzy ; Tomaszewski, Maciej ; Döring, Angela ; Peters, Annette ; Wichmann, H.-Erich ; Kaess, Bernhard M. ; Kalbitzer, Hans Robert ; Huber, Fritz ; Pfahlert, Volker ; Samani, Nilesh J. ; Kronenberg, Florian ; Dieplinger, Hans ; Illig, Thomas ; Hengstenberg, Christian ; Suhre, Karsten ; Gieger, Christian ; Kastenmüller, Gabi
; Römisch-Margl, Werner ; Kremer, Werner ; Raffler, Johannes ; Krug, Susanne ; Skurk, Thomas ; Rist, Manuela J. ; Daniel, Hannelore ; Hauner, Hans ; Adamski, Jerzy ; Tomaszewski, Maciej ; Döring, Angela ; Peters, Annette ; Wichmann, H.-Erich ; Kaess, Bernhard M. ; Kalbitzer, Hans Robert ; Huber, Fritz ; Pfahlert, Volker ; Samani, Nilesh J. ; Kronenberg, Florian ; Dieplinger, Hans ; Illig, Thomas ; Hengstenberg, Christian ; Suhre, Karsten ; Gieger, Christian ; Kastenmüller, Gabi 
Zusammenfassung
Adverse levels of lipoproteins are highly heritable and constitute risk factors for cardiovascular outcomes. Hitherto, genome-wide association studies revealed 95 lipid-associated loci. However, due to the small effect sizes of these associations large sample numbers (100 000 samples) were needed. Here we show that analyzing more refined lipid phenotypes, namely lipoprotein subfractions, can ...
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