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Publikationen von 0000-0001-7927-858X
(ORCID: 0000-0001-7927-858X)

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Gutiérrez, A., Scharl, M., Sempere, L. , Holler, Ernst, Zapater, P. , Almenta, I., González-Navajas, J. M. , Such, J., Wiest, Reiner, make_name_string expected hash reference und make_name_string expected hash reference (2014) Genetic susceptibility to increased bacterial translocation influences the response to biological therapy in patients with Crohn's disease. Gut 63, S. 272-280.

Hellerbrand, Claus, make_name_string expected hash reference, make_name_string expected hash reference , make_name_string expected hash reference, make_name_string expected hash reference , make_name_string expected hash reference, make_name_string expected hash reference , make_name_string expected hash reference, make_name_string expected hash reference, make_name_string expected hash reference und make_name_string expected hash reference (2014) Molecular targets for antifibrotic therapy in liver disease: using magic bullets for crossfire rather than a one-sided shotgun attack. Gut 63, S. 1039-1041.

Gutiérrez, Ana, Scharl, Michael, Sempere, Laura, Holler, Ernst, Zapater, Pedro , Almenta, Isabel, González-Navajas, José, Such, José, Wiest, Reiner, Rogler, Gerhard und Francés, Rubén (2012) Intensified Anti-TNF-Alpha Therapy and Bacterial-DNA Translocation in Patients With Mutated NOD2/ATG16L1-Combined Genotypes. Inflammatory Bowel Diseases 18, S60. Volltext nicht vorhanden.

Gutiérrez, Ana, Holler, Ernst, Zapater, Pedro , Sempere, Laura , Jover, Rodrigo, Pérez-Mateo, Miguel, Schoelmerich, Juergen, Such, José, Wiest, Reiner und Francés, Rubén (2011) Antimicrobial peptide response to blood translocation of bacterial DNA in Crohnʼs disease is affected by NOD2/CARD15 genotype. Inflammatory Bowel Diseases 17 (8), S. 1641-1650. Volltext nicht vorhanden.

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