Einträge von Tenesa, Albert auf dem Publikationsserver

Grosche, Sarah, Marenholz, Ingo, Esparza-Gordillo, Jorge, Arnau-Soler, Aleix, Pairo-Castineira, Erola , Rüschendorf, Franz, Ahluwalia, Tarunveer S. , Almqvist, Catarina, Arnold, Andreas, Baurecht, Hansjörg , Bisgaard, Hans , Bønnelykke, Klaus , Brown, Sara J., Bustamante, Mariona, Curtin, John A., Custovic, Adnan, Dharmage, Shyamali C., Esplugues, Ana, Falchi, Mario , Fernandez-Orth, Dietmar, Ferreira, Manuel A. R., Franke, Andre , Gerdes, Sascha , Gieger, Christian, Hakonarson, Hakon, Holt, Patrick G. , Homuth, Georg, Hubner, Norbert, Hysi, Pirro G., Jarvelin, Marjo-Riitta, Karlsson, Robert, Koppelman, Gerard H. , Lau, Susanne, Lutz, Manuel, Magnusson, Patrik K. E., Marks, Guy B., Müller-Nurasyid, Martina, Nöthen, Markus M., Paternoster, Lavinia, Pennell, Craig E., Peters, Annette , Rawlik, Konrad, Robertson, Colin F., Rodriguez, Elke, Sebert, Sylvain, Simpson, Angela , Sleiman, Patrick M. A., Standl, Marie, Stölzl, Dora, Strauch, Konstantin, Szwajda, Agnieszka, Tenesa, Albert, Thompson, Philip J., Ullemar, Vilhelmina, Visconti, Alessia, Vonk, Judith M., Wang, Carol A., Weidinger, Stephan, Wielscher, Matthias , Worth, Catherine L., Xu, Chen-Jian und Lee, Young-Ae (2021) Rare variant analysis in eczema identifies exonic variants in DUSP1, NOTCH4 and SLC9A4. Nature Communications 12 (1). Volltext nicht vorhanden.

Bergmann, Sven , Baillie, J. Kenneth , Bretherick, Andrew, Haley, Christopher S. , Clohisey, Sara, Gray, Alan, Neyton, Lucile P. A., Barrett, Jeffrey, Stahl, Eli A., Tenesa, Albert , Andersson, Robin, Brown, J. Ben , Faulkner, Geoffrey J., Lizio, Marina, Schaefer, Ulf , Daub, Carsten , Itoh, Masayoshi, Kondo, Naoto , Lassmann, Timo, Kawai, Jun, Mole, Damian , Bajic, Vladimir B., Heutink, Peter, Rehli, Michael, Kawaji, Hideya , Sandelin, Albin, Suzuki, Harukazu, Satsangi, Jack , Wells, Christine A., Hacohen, Nir , Freeman, Thomas C., Hayashizaki, Yoshihide , Carninci, Piero , Forrest, Alistair R. R. und Hume, David A. (2018) Shared activity patterns arising at genetic susceptibility loci reveal underlying genomic and cellular architecture of human disease. PLOS Computational Biology 14 (3), e1005934. Volltext nicht vorhanden.