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- URN zum Zitieren dieses Dokuments:
- urn:nbn:de:bvb:355-epub-305265
- DOI zum Zitieren dieses Dokuments:
- 10.5283/epub.30526
Zusammenfassung
Human cancers almost ubiquitously harbor epigenetic alterations. Although such alterations in epigenetic marks, including DNA methylation, are potentially heritable, they can also be dynamically altered. Given this potential for plasticity, the degree to which epigenetic changes can be subject to selection and act as drivers of neoplasia has been questioned. We carried out genome-scale analyses ...
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