UR Home
![]() | Up a level |
(2020)
Oxidative stress modulates rearrangement of endoplasmic reticulum-mitochondria contacts and calcium dysregulation in a Friedreich's ataxia model.
Redox Biology 37, p. 101762.
Fulltext not available.
Edenharter, Oliver, Schneuwly, Stephan and Navarro, Juan A.
(2018)
Mitofusin-Dependent ER Stress Triggers Glial Dysfunction and Nervous System Degeneration in a Drosophila Model of Friedreich’s Ataxia.
Frontiers in Molecular Neuroscience 11 (38), pp. 1-25.
Navarro, Juan A. and Schneuwly, Stephan
(2017)
Copper and Zinc Homeostasis: Lessons from Drosophila melanogaster.
Frontiers in Genetics 8 (223), pp. 1-20.
, Lind, Maria I. and Schneuwly, Stephan
(2015)
Mitoferrin modulates iron toxicity in a Drosophila model of Friedreich׳s ataxia.
Free Radical Biology and Medicine 85, pp. 71-82.
Fulltext not available.
, Llorens, José V., Soriano, Sirena, Botella, José A., Schneuwly, Stephan, Martínez-Sebastián, María J.
and Moltó, María D.
(2011)
Overexpression of Human and Fly Frataxins in Drosophila Provokes Deleterious Effects at Biochemical, Physiological and Developmental Levels.
PLoS ONE 6 (7), e21017.
Fulltext not available.
, Navarro, Juan A., Martínez‐Sebastián, Maria J., Baylies, Mary K., Schneuwly, S., Botella, José A. and Moltó, Maria D.
(2007)
Causative role of oxidative stress in a Drosophila model of Friedreich ataxia.
The FASEB Journal 21 (2), pp. 333-344.
Fulltext not available.
Publication Server
Publishing: oa@ur.de
0941 943 -4239 or -69394
Dissertations: dissertationen@ur.de
0941 943 -3904
Research data: datahub@ur.de
0941 943 -5707